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ERK activation leads to various disease conditions by promoting collagen synthesis resulting in cardiac fibrosis, disruption of mitochondrial activities by reducing the synthesis of ATP and releasing ROS, upregulating ER stress, the activation of JNK signalling and altering the signal cascades related to myocardial dysfunction resulting in DCM

The patients glucose and blood pressure remained stable without medication

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Systemic adverse effects and toxicities associated with immunotherapy: A review