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As intracellular GSH is exhausted, the GSH synthesis system will be activated accordingly, which would weaken the efficacy of GSH exhaustion-based cancer therapies in long-term treatment (Xiong et al., 2021)

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Therapeutic administration of either progranulin or soluble versions of TNFR1 or PLAD in mouse models led to beneficial results [189,190,191], opening the door to another type of non-antibody anti-TNF - drugs [23, 192]