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Neuroprotective actions of taurine on hypoxic-ischemic brain damage in neonatal rats
When it crosses the blood-brain barrier and binds to c-Met, it initiates synaptogenesis: new dendritic spines form, synaptic connections strengthen, and neurons that were alive but under-connected begin to integrate more fully into functional networks
Plasma and tissue GSH levels decline with age, while markers of oxidative damage increase, creating a "redox imbalance" implicated in the pathogenesis of age-related diseases [2][3][10][11]
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