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In moderate to severe TBI the signaling pathways become more complicated, and there are many potential pathways that can be targeted and blocked for therapeutic intervention like IL-6, TNFa, TLRs, S100 (DAMPs), RAGE, JAK2/STAT3, and/or VEGF/MET to prevent neuroglial overactivation, neuroinflammation, gliosis, loss of inhibition, synaptic knock-down, cell death, and chemokine secretion, and we hope to conduct further research on these conditions (2a)
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5d and Extended Data Fig
Comorbid conditions such as ADHD, depression, and anxiety disorder frequently co-occur in individuals with ASD in clinical settings [65, 66], and their inclusion in the analysis would yield results more reflective of real-world conditions for these individuals
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