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In the adult male SE offspring, reduced mitochondrial MnSOD suggests increased oxidative stress which may be linked to increased OXPHOS complex III, which is the major site of ROS production
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Bellar, D., LeBlanc, N
When it binds MC1R on melanocytes, it triggers cyclic adenosine monophosphate (cAMP) signaling, which upregulates tyrosinase activity and accelerates melanogenesis (i.e
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