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Relying on a single biomarker, whether derived from neuroimaging, biofluids, electrophysiology, or clinical scales, has proven insufficient to capture the complexity of neuronal injury and recovery [586, 587]
We observed that AOAA treatment decreases cysteine and GSH levels while increases ROS and ferroptosis stresses in SCD erythroblasts (Supplementary Fig
It showed excellent safety with no serious adverse events and no IGF-1 elevation
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