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15 , mjad064 (2024)
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tb was determined within in vitro granulomas of infected-PBMCs from T2DM subjects that were either untreated (control), treated in vitro with PZA (one-time addition at 5 g/ml) or with INH (one-time addition at 0.0125 g/ml)
Chronic hyperglycemia initiates a cascade of molecular eventsincluding the accumulation of advanced glycation end products (AGEs), activation of the polyol pathway, enhanced protein kinase C (PKC) signaling, and mitochondrial dysfunctionculminating in glomerular hyperfiltration, podocyte injury, and progressive glomerular and tubulointerstitial fibrosis
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