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The rationale for combining them is mechanistic complementarity (different signaling pathways, both implicated in tissue repair) and clinical anecdote in the peptide-therapy community not formal head-to-head or combination-vs-monotherapy data
Conventional drugs show limitations in neurological disorders, especially brain tumors, as oral administration leads to hepatic metabolism and reduced concentration at the target (Tang et al
Deficiency of cytosolic arylamine N-acetylation in the domestic cat and wild felids caused by the presence of a single NAT1-like gene
These observations show that individual GST genes/enzymes may suppress plant resistance in the initial biotrophic phase of the infection, possibly by providing a high antioxidative capacity favorable to the fungus (Hernndez et al., 2009)
