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Direct effects on cellular proliferation A genetically engineered murine model of pancreatic intraepithelial neoplasia (PanIN) to PDAC progression ( LSL-Kras G12D ;Pdx1-Cre, KC ) treated with exenatide demonstrated architectural changes of loss of polarity and micropapillary formation following 12 weeks of therapy 18

Mandlekar and Kong, 2001), was also significantly attenuated by GSTP1 (Supplementary Figure S4), which cannot be explained by GSTP1-mediated inactivation of reactive metabolites in the absence of bioactivation of tamoxifen in HepG2 cells

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