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In Alzheimers disease models, riboflavin suppresses p38 MAPK and NF-B activation, reducing amyloid--induced neuroinflammation and microgliosis ( Together, these findings underscore riboflavins dual neuroprotective role: inhibiting pro-inflammatory signaling at the cellular level, and reinforcing BBB stability to reduce neuroinflammatory propagation

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Glucose depletion enhances P-glycoprotein expression in hepatoma cells: role of endoplasmic reticulum stress response

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