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Site-directed mutagenesis and inhibition assays revealed that Phe-9 is potentially an important residue for the binding of the inhibitor S-hexyl glutathione (GTX) [77]
15:e0228568
Together with other lipid-soluble antioxidants, these pathways suppress membrane lipid peroxidation and preserve membrane integrity, thereby preventing ferroptotic cell death (Doll et al., 2019
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