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In theory, an increase in the cytosolic Ca 2+ concentration of an erythrocyte may be the consequence of Ca 2+ influx from the extracellular space, decreased Ca 2+ transport from the cytosol to the extracellular space, or release from intracellular Ca 2+ binders
Meanwhile, through SIRT1-mediated NRF2 deacetylation, VD enhances HO-1 expression, facilitating heme degradation and bilirubin production [61]
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