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Collectively, GGCT functions as a metabolic rheostat that orchestrates mitochondrial homeostasis through diverting Gln flux toward glutathione biosynthesis to fortify redox defense, while enhancing Glc-fueled TCA cycle anaplerosis to sustain cell proliferation

Despite this preclinical evidence, our pathway analysis reveals that the Neuroactive Ligand-Receptor Interaction Pathway does not directly integrate into the DM2-AD comorbidity network, supporting limited engagement with core diabetes-associated neurodegenerative mechanisms

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PMID: 37 MayoKEMillerLJBatailleDDalleSGkeBThorensBet al