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*Reduction in A1C is dependent upon a number of factors, such as baseline A1C and background therapy
That's the part of the GLP-1 conversation that doesn't get enough airtime
Experimental and human metabolic literature demonstrates that GH promotes fatty acid mobilization, shifts substrate preference toward fat oxidation, and alters carbohydrate-lipid partitioning during fasting and metabolic stress states, making GH secretagogue systems useful tools for investigating adipose tissue signaling, fuel selection, and the endocrine regulation of energy metabolism [8]
Timeline: High-dose results won't be available until 2028+, well after standard-dose approval
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