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Clinically, this pathological hyperplasia often manifests as bladder outlet obstruction and LUTS, with severe cases potentially resulting in chronic renal failure and cardiovascular complications [5]

At a final magnification of 770 , a stage micrometer scanned equidistantly along the X and Y axes, examining 8 to 14 tiny fields for each liver sample

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Similarly, patients with factor XII (FXII) deficiency do not have a bleeding tendency and remain practically asymptomatic, although its dysregulation has been associated with hereditary angioedema, thrombosis, and neuroinflammation [8]