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Koch CE, Leinweber B, Drengberg BC, Blaum C, Oster H

K Ca channels respond to signals from activated MRs, facilitating K + influx

After being activated by insulin signaling, Akt inactivates AS160 (GTPase-activating protein [GAP] AKT substrate of 160 kDa, also known as TBC1D4), which activates small Rab GTPase protein switches that control vesicle trafficking [10]

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