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Br J Clin Pharmacol 2012;74:437444
10.1016/j.gene.2013.10.071 Gene 34 KohC
These interactions can modulate immune responses, reduce inflammatory signaling, strengthen tight junctions between intestinal cells (reducing permeability), and potentially support the environment in which L-cells function optimally
Whether these safety signals translate equivalently to MC4R -deficient patients - who are predominantly children and adolescents with a distinct metabolic phenotype - remains unknown
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