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This genetic condition results from the presence of an extra copy of chromosome 21, as first described by APP ) (Salehi et al., 2007), the Glutamate Receptor, Ionotropic, Kainate 1 ( GRIK1 ) (Valbuena et al., 2019), the Regulator of CAlciNeurin 1 ( RCAN1 ) ( Dual-specificity tyrosine phosphorylation-Regulated Kinase 1A ( DYRK1A ) (Nakano-Kobayashi et al., 2017

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Stoner, M