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It is well established that patients with diabetes demonstrate higher susceptibility to pancreatic cancer and pancreatitis compared to non-diabetic individuals, and theoretically, sustained GLP-1 receptor activation induced by GLP-1RA therapy may potentially elevate pancreatic cancer risk [8]

Our metagenomic data revealed a SE-driven shift away from a pro-inflammatory dysbiotic statecharacterized by the depletion of Bacteroides and enrichment of Desulfovibrio toward a neuroprotective microbial signature