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Their cost-effectiveness and favorable pharmacokinetic properties (e.g., efficient absorption and potent bioactivity) make them attractive candidates for localized and targeted wound therapies [29, 54-57]
In colitis, multiple proinflammatory cytokines within the intestinal tissue abnormally activate STAT3, resulting in intestinal barrier dysfunction, hyperactivation of innate immunity, and enhanced Th17-mediated immune responses, all of which contribute to chronic intestinal inflammation (Chen X
Subsequently, p62 recruits ubiquitinated labeled mitochondria to autophagosomes by binding to LC3 and eventually fuses with lysosomes to form autolysosomes that remove damaged mitochondria
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