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This study aims to explore the link between endothelial dysfunction, a key component of HFpEF, and the therapeutic actions of GLP-1RAs
An in vitro study in hCAECs found that exenatide decreased inflammation by reducing activation of nuclear factor kappa-light-chain-enhancer of activated B cells (NF-B), adhesion molecules intracellular adhesion molecules (ICAM) and vascular cell adhesion molecules (VCAM), but not P-selectin ( Third, GLP-1 RAs potentially affect coronary plaque characteristics
Mouse monoclonal anti-GAPDH antibody (1:1000
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