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National Library of Medicine, Accessed 20 Aug
However, most experts agree that this is well below the physiological needs of most individuals
Mitochondria-derived peptide MOTS-c alleviates hyperoxia-induced bronchopulmonary dysplasia in neonatal mice by activating Nrf2 pathway
[19] [1] This washout period allows for adequate drug clearance and minimizes potential fetal exposure during early pregnancy
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