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Combining the therapeutic approaches of GLP-1 agonists (which target extrahepatic mechanisms for metabolic disorders) with resmetirom (which target intrahepatic mechanisms for MASH and liver fibrosis) seems like a promising strategy for addressing fat accumulation, inflammation, and fibrosis associated with MASH

Such modifications also reduce enzymatic degradation rates, thereby prolonging the metabolic half-life of flavonoids within the body (Lee et al., 2016)

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