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IGF-1 induces muscle hypertrophy through satellite cell activation and increased protein synthesis in differentiated myofibers (Adams & McCue, 1998)
However, it is essential to note that while the peptide appears to elevate the expression of beta(3)-AR, it may not act directly through the beta(3)-AR to induce its potential lipolytic action
This suggests that GLP-1 and insulin secretions have striking commonalities in the synergistic release and signaling mechanisms
Overall, these differences underscore the importance of dataset harmonization and the inclusion of larger, more diverse cohorts in future studies
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