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Similar findings were obtained in a human cisplatin-resistant cervix squamous cancer cell line (Pandit et al., 2021)
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Animal studies have further demonstrated that ApoE4 influences -synuclein pathology and exacerbates its detrimental effects, suggesting a role in impairing -synuclein clearance and promoting neuroinflammation (Zhao et al., 2020
In conclusion, this study not only identifies Sema as a potential therapeutic agent for pathological myocardial remodeling but also suggests NR4a1 as a key target for regulating the myocardial glucolipid metabolic pathway
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