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Persistent activation of GLP-1 receptors in exocrine pancreatic cells may lead to localized hyperplasia in the exocrine pancreas, consequently accelerating the formation of dysplastic lesions and pancreatitis [64]
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Results obtained in preclinical models indicate that GLP-1 improves endothelial dysfunction, prevents vascular remodeling, and activates the endothelial nitric oxide synthase-nitric oxide-cGMP pathway
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