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pylori evolved a series of virulence factors to ensure its survival and pathogenicity [37]
These may include headaches, dizziness, and gastrointestinal issues like nausea or stomach pain
For conditions where runaway inflammation is the dominant pathology ulcerative colitis at a cellular level, for example BPC-157 alone may not fully address the inflammatory mechanism
(iv) Gallbladder stasis plus mucin hypersecretion and mucin gel accumulation in the gallbladder lumen, as well as immune-mediated gallbladder inflammation contribute to the pathogenesis of cholesterol gallstones
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