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The value of GSH as an immunopharmacologic agent is that it can finetune immune response in a disease-dependent and redox state-dependent manner which differs from typical broad-effect immunosuppressive treatments (Diotallevi et al., 2017) (Figure 3)
445,447 Overall, the selection of proper drug treatments for nonmotor symptoms should rely on the efficacy and extent of adverse events
GHK-Cu also modulates gene expression through mechanisms that remain incompletely characterized but involve broad transcriptomic changes affecting over 4,000 human genes as identified through Connectivity Map (CMAP) analysis
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