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The liquid solution is far more fragile, and a new set of rules applies
Thyroglobulin type-I-like domains in invariant chain fusion proteins mediate resistance to cathepsin L digestion
project administration and funding acquisition: N.I.M.F
Proposed Mechanism BPC-157 appears to work through multiple pathways: Maintaining vesicular integrity (affecting VMATs and dopamine transporters) Possibly occupying its own vesicular storage sites Competing for dopamine receptor binding Functionally substituting when dopaminergic neurons are lost This last property, essentially filling in for missing dopamine function, would explain why the peptide counteracts both receptor agonists (apomorphine) and antagonists (haloperidol, fluphenazine, clozapine, sulpiride) with equal effectiveness
