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The urea level ( Figure 7C ) in CAR was on average 11% higher than in CON throughout the study (p G = 0.029)

In the context of white adipogenic differentiation, commonly impaired in T2D, semaglutide increased adipocyte numbers and induced a shift toward a more lipolytic phenotype, as indicated by a reduction in medium-sized lipid droplets

e.g., angiotensin-converting enzyme inhibitor(s) [ACEi], or angiotensin receptor blockers [ARB]), currently available treatments include sodium-glucose cotransporter 2 inhibitors (SGLT2i), glucagon-like peptide-1 receptor agonists (GLP-1 RA), and nonsteroidal mineralocorticoid receptor antagonists (ns-MRA), such as finerenone [5,6,7,8]

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