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Hyperuriceamia may aggravate kidney damage through RAS activation which is an important mediator of kidney disease progression or through developing hypertension by increasing salt sensitivity (Kang et al., 2002
Beighton, P., Solomon, L
The proposed logic is that glutathione acts as a cellular antioxidant reducing oxidative stress, while the GLP-1 medication drives weight loss and metabolic improvement, and that lowering systemic inflammation might amplify the metabolic result
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