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Dietary intake is modest estimated at 100-150 mg/day from fruits, vegetables, and meats and much of this is broken down by digestive enzymes (gamma-glutamyltranspeptidase and dipeptidases) in the intestinal lumen before absorption [1][2]
(2020) specifically addressed NSAID-induced gut damage a clinically relevant injury model and documented that BPC-157 administration stabilized the claudin-4 and occludin tight junction proteins that are degraded by NSAID exposure, providing a mechanistic explanation for the peptides permeability-protective effects
10.1016/j.phrs.2005.12.005
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