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Neurons pretreated with Dihexa demonstrate significantly enhanced survival when exposed to reactive oxygen species, excitotoxic glutamate concentrations, or metabolic impairment compared to untreated controls
Although this finding was consistent with the perspective that cell-autonomous HIF1a in Tbr2 + INPs caused oxidative stress among neural progenitors, HIF1a is also normally present in multiple cell types throughout the SGZ where it acts as a pro-neurogenesis signal among early neuroprogenitors 48,61
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