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Key limitation: Most injectable GHK-Cu dosage protocols are extrapolated from topical human trials and animal studies

HDAC3 and HDAC11, for instance, regulate oxidative stress responses and A metabolism, whereas HDAC7 contributes to tau pathology by suppressing lysosomal biogenesis through transcription factor EB (TFEB) deacetylation and by enhancing inflammatory signaling via the IKKNF-B pathway 31

Continuous, multi-month use is widely discouraged because of progressive insulin resistance, organ-growth concerns, and IGF-1 receptor desensitization

It remains a subject of ongoing scientific evaluation in controlled research environments