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These bioactive compounds may modulate key signaling pathways, including mTOR, NF-B, and AMPK, while also influencing epigenetic processes such as DNA methylation, histone modifications, and microRNA regulation

In summary, through pan-cancer analysis using the TIMER database combined with mechanistic integration, this review demonstrates that the activation patterns of the serine synthesis pathway (SSP) in malignant tumors are not randomly distributed, but follow three distinct molecular subtypes: driver mutation-dependent, microenvironmental stress-adaptive, and epigenetic dysregulation subtypes

Their potential effects overlap, and the clinical evidence for both remains limited

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