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Insulin/IGF signaling and discoidin domain receptors: an emerging functional connection
Our analyses demonstrated that PD patients exhibit significant metabolic reprogramming, characterized by a shift in energy metabolism from the tricarboxylic acid cycle toward glycolysis, a dysregulated urea cycle, and lipid remodeling, as well as extensive activation of inflammatory and immune responses involving the PI3K-Akt, IL-17, NF-kappaB, MAPK and TNF signaling pathways
SiO(2)-induced ferroptosis in macrophages promotes the development of pulmonary fibrosis in silicosis models
Varinthra P, Liu IY
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