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Moreover, in agreement with the critical role of the capsule for meningococcal intracellular survival (Spinosa et al., 2007), siaD deletion in the MC58 strain led to an increase in adhesion to Calu3 LLI but to a strong reduction in intracellular bacteria and in transcellular crossing (Sutherland et al., 2010)

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Recently, RNAi-mediated knockdown of four overexpressed GST genes in the imidacloprid resistant N