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It was designed to have a much longer half-life in circulation compared to natural IGF-1
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Further, NRF1 stimulates the expression of mitochondrial transcription factor A (TFAM), which is used for the duplication of mitochondrial DNA [83]
Nitric oxide can directly stimulate augmented synthesis and secretion of corticosterone by NO-COX-Prostaglandin pathway and enhance hypoxia induced neurodegeneration [17]
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