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Although these preclinical findings are promising for muscle quality, they need to be confirmed in clinical studies, as human data remain scarce
R.DaltonA
Our findings uncover an extra-organellar buffering mechanism whereby partial v-ATPase inhibition mitigates mitochondrial dysfunction by altering pH homeostasis and driving metabolic rewiring as a protective response that promotes cell fitness
The gene expressions of RBCS , sedoheptulose-1,7-bisphosphatase ( SBPase ), and FBPase were significantly increased under the NT+GSH treatment compared to the NT treatment
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