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Exp Neurol 203(2):51220, PMID: 17049515 Shen H, Liu Z (2006) JNK signaling pathway is a key modulator in cell death mediated by reactive oxygen and nitrogen species

It should be emphasized that this effect does not necessarily mean that Asp itself is a pro-inflammatory factor, but more likely reflects its position in the coupling of energy metabolism and metabolic signaling: when shuttle activity and anabolic demand increase, Asp supply may become one of the limiting factors supporting pro-inflammatory transcription and effector molecule synthesis

Specifications Research-grade material supplied in lyophilized vial presentation at 10mg

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