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Homology modeling reveals that these mutations create a more expansive substrate-binding pocket, optimally shaped to accommodate short-chain (C3C5) methionine homologs and distinguishing it from its paralog, MAM3, which specializes in longer-chain (C6C8) substrates (20)

The connection between obesity and CVD lies in dysfunctional adipose tissue, which releases various harmful bioactive lipids and pro-inflammatory factors (known as adipokines) that negatively affect the cardiovascular system