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Furthermore, during the normal aging processes, the ability of cells to maintain their normal redox state diminishes (Go and Jones, 2017

Cimino JA et al

Its primary mechanism is increasing VEGF expression (vascular endothelial growth factor), which promotes new blood vessel growth at injury sites

In vitro assays using BaF-BO3 cells transfected with the hGH receptor confirmed that AOD9604 does not compete for the hGH receptor and does not induce cell proliferation unlike intact hGH establishing that it operates via a novel mechanism distinct from traditional hGH-stimulated pathways