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This resistance allows cancer cells to tolerate ROS accumulation and maintain survival unless the balance is further disrupted by external ROS inducers (e.g., H 2 O 2 ) or inhibitors of ferroptosis-resistance mechanisms (e.g., RSL3 and Erastin)
doi: 10.1530/eje.0.1330425 128 FrieboesMurckAntonijevicSteiger
Hegazy UM, Mannervik B, Stenberg G
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