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Meanwhile, LSN3318839, an orally effective positive allosteric modulator selective for GLP-1(9-36), anchors in the TM1-TM2 cleft and repositions GLP-1(9-36) to adopt a binding pose like GLP-1(7-36), thereby enhancing GLP-1R-mediated signal transduction
The authors propound that deficient islet GABA signaling/content may contribute to the hyper-glucagonemia of T2D which again reinforces a role for GABA-based therapeutics
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