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Larger particles, by contrast, benefit from prolonged circulation and enhanced accumulation at the tumor periphery, but their restricted penetration into deeper tissue regions remains a persistent limitation [33, 38, 40]

Too much glutamate induces excessive stimulation of glutamate receptors and increases the concentration of Na + and Ca 2+ in the cell, which may directly cause neuronal damage and cell death

GHK exhibits a Cu 2+ affinity comparable to that of albumins copper transport sites, forming an in vivo complex, glycyl-L-histidyl-L-lysine-Cu 2+ (GHK-Cu), which enhances its bioavailability (Pickart, 2008)

GHK-Cu is typically administered subcutaneously in cycles, with the average length of use running around 8 weeks followed by a rest period