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How TB-500 promotes regeneration The primary mechanism involves actin polymerization
PMID 8292944
Nrf2 depletion in macrophages leads to increased foam cell formation, increases the M1 inflammatory phenotype with enhanced expression of pro-inflammatory monocyte chemoattractant protein-1 and interleukin-6, and aggravates atherosclerosis (244, 245)
This is why cagrilintide, which operates through completely different receptors, makes a more logical combination partner than additional GLP-1 agonists
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