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Reports indicate attenuation of rhythm disturbances and reduced thrombus formation, pointing toward a broad spectrum of cardiovascular protective activity
This is just one of several pathways by which GHK-Cu is thought to work
In preclinical models, BPC157 has been shown to modulate expression of key inflammatory mediators by downregulating proinflammatory transcription factors such as NFB and AP1 while upregulating antiinflammatory genes linked to IL10 signaling, thereby shifting the balance toward resolution of inflammation in chronically injured tissues (24,25)
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