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Overall, glutathiones neuroprotective efficacy in the CNS is intricately regulated by enzyme expression, metabolic substrate availability, and regional brain biochemistry, making it a pivotal target for intervention in oxidative stress-related neuropathologies

Early infection or trauma and schizophrenia This may seem odd or even frightening that schizophrenia can result from the whims of a virus or bacteria

A common pathway affecting all these risk factors involves a low measure of reduced glutathione (i.e., GSH) level (Khanfar and Al Qaroot, 2020

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