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Further mechanistic investigations have implicated GHK-Cu in the activation of the Nrf2/ARE antioxidant response pathway in cultured cells, where it appears to reduce reactive oxygen species (ROS) burden independently of direct radical scavengingsuggesting an indirect, gene-regulatory mode of action rather than purely stoichiometric antioxidant chemistry

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The combination of powerful antioxidants, which inhibit the destructive effects of ultraviolet light, free radicals and other environmental factors, promotes the active restoration of damaged cells