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GSH depletion does not induce mitochondrial superoxide or alter mitochondrial mass in RPE cells It has been suggested that mitochondria are predominant source of ROS upon H 2 O 2 -induced oxidative stress 7

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2.3 Free radical scavenging capacity of Ru SA+AC on oxidative stress rat chondrocytes in vitro An overabundance of RONS can lead to increased expression of inflammatory factors, mitochondrial impairment, and the induction of cell apoptosis, thereby accelerating the progression of OA[64]

Consult physician if pregnant/nursing, taking medication, or have a medical condition (especially glaucoma)